CEPROTIN - Replacement Therapy for Protein C Deficiency

In the human, blood normally coagulates within seconds of an injury or forms blood clots (platelets (tiny cells in the blood) congregate around ruptures in blood vessels to form a clot, through a complex process involving as many as twenty different plasma proteins (manufactured in the liver), known as coagulation factors. Plasma proteins C and S inhibit (anticoagulants -- prevent blood clots) reverse the process to prevent excessive clotting. Major structural component of blood clots is Fibrin fiber, creates a fibrin mesh. The fibrin mesh is stronger and more stable then the temporary platelet scab. Days after platelets form a clot, fibrin mesh strengths even more, protecting the blood vessels from further damage or blood loss. A protein called plasma is formed that dissolves fibrin (after the injury heals) and the blood clot is slowly removed.